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Fun Facts About Flu Vaccinations
Increased
Risk of Noninfluenza Respiratory Virus Infections Associated with
Receipt of Inactivated Influenza Vaccine Vaccination
may make flu worse if exposed to a second strain Flu shot
shedding: “In adjusted models, we observed 6.3 (95% CI 1.9–21.5)
times more aerosol shedding among cases with vaccination in the
current and previous season compared with having no vaccination
in those two seasons.” Shedders
are disease spreaders. Some recipients of the influenza vaccine
can shed the disease for up to 4 weeks following vaccination,
depending on the vaccine. Up to 89% of vaccinated infants shed
the live virus. Recently vaccinated teenagers and adults shed
and spread the influenza virus as well. Source: vaccine manufacturer's
product insert, table 5:
Vaccinated people at higher risk for illness https://www.ncbi.nlm.nih.gov/pubmed/?term=24483149 https://www.ncbi.nlm.nih.gov/pubmed/?term=29348203 https://www.ncbi.nlm.nih.gov/pubmed/?term=22423139 https://www.ncbi.nlm.nih.gov/pubmed/?term=21079528 https://www.ncbi.nlm.nih.gov/pubmed/?term=25844934 https://www.ncbi.nlm.nih.gov/pubmed/?term=21880755 https://www.sciencedaily.com/releases/2009/05/090519172045.htm
Spreading of disease by vaccine recipients "Presymptomatic
shedding may arise in one third of cases" Asymptomatic
spreading:
Flu shot
failure
Non-specific
immunity to other infections is destroyed by the flu vaccine
Stroke
following influenza vaccination
Autoimmunity
following vaccination
Observe
two very provaccine scientists who say that the risk of developing
CNS demyelination following vaccination in general is “low
but not non negligible.”
"Although prevention of influenza virus infection is the desirable outcome after influenza vaccination, people may still become infected with influenza despite being vaccinated." – Osterholm MT, Kelley NS, Sommer A, Belongia EA. Efficacy and effectiveness of influenza vaccines: a systematic review and meta-analysis. Lancet Infect Dis 2012; 12:36-44
Ethyl vs. Methyl mercury "debate" And for those pro-vaccine folks who are still spewing the ethyl v. methyl nonsense, consider that there is no significant difference in human toxicity as: Both ethyl and methyl mercury cause DNA damage or impair DNA synthesis (Burke et al. 2006; Sharpe et al. 2012; Wu et al. 2008). Both cause oxidative stress/creation of reactive oxygen species (Dreiem and Seegal 2007; Garg and Chang 2006; Myhre et al. 2003; Sharpe et al. 2012; Yin et al. 2007). Both decrease glutathione activity, thus providing less protection from the oxidative stress caused by MeHg and EtHg (Carocci et al. 2014; Ndountse and Chan (2008); Choi et al. 1996; Franco et al. 2006; Mori et al. 2007; Muller et al. 2001; Ndountse and Chan 2008; Wu et al. 2008). Both cause effects on cell division by damaging the spindle apparatus during mitosis (Burke et al. 2006; Castoldi et al. 2000; Gribble et al. 2005; Kim et al. 2007; Ou et al. 1999b; Machaty et al. 1999; Rodier et al. 1984). Both MeHg and EtHg bind to the amino acid cysteine (Clarkson 1995; Wu et al. 2008). Both MeHg and EtHg strongly inhibit the reacylation of arachidonic acid, thus inhibiting the reincorporation of this fatty acid into membrane phospholipids (Shanker et al. 2002; Verity et al. 1994; Zarini et al. 2006). Both cause an increase in NOS, causing an overproduction of NO (Chen et al. 2003; Chuu et al. 2001; Shinyashiki et al. 1998). Both disrupt glutamate homeostasis (Farina et al. 2003a, b; Manfroi et al. 2004; Mutkus et al. 2005; Yin et al. 2007). Both alter intracellular calcium homeostasis (Elferink 1999; Hare et al. 1993;Kang et al. 2006; Limke et al. 2004b; Machaty et al. 1999; Marty and Atchison1997; Minnema et al. 1987; Peng et al. 2002; Sayers et al. 1993; Sirois and Atchison, 2000; Szalai et al. 1999; Tornquist et al. 1999; Zarini et al. 2006). Both cause effects on receptor binding/neurotransmitter release involving one or more transmitters (Basu et al. 2008; Coccini et al. 2000; Cooper et al. 2003; Fonfria et al. 2001; Ida-Eto et al. 2011; Ndountse and Chan 2008; Yuan and Atchison 2003).
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